COMMUNICABILITY: There is no documented evidence of person-to-person transmission of VSV.
SECTION III-DISTRIBUTION
RESERVOIR: The main reservoir is the sand fly, although arboreal rodents and nonhuman primates can also harbor VSV (7). Grasshoppers have also been implicated as a potential reservoir for VSV (4).
ZOONOSIS: Yes, humans can contract VSV through direct contact with infected animals, or indirectly through the bite of an infected fly(1,5,7,8).
VECTORS: Sand flies (Phlebotomus spp.) appear to be is telegram available in south korea the most important vector for VSV(2,6,8). Black flies (Simuliidae) (2,5,6), mosquitoes (Culicoides spp.), midges (Aedes spp.)(2,5,8) and other Dipterans(2,5,6) have also been implicated.
SECTION IV – STABILITY AND VIABILITY
DRUG SUSCEPTIBILITY: Unknown.
SENSITIVITY TO DISINFECTANTS: VSV is inactivated by 1% cresylic acid, phenolics, chlorinated phenol, 2.5% phenol, 0.4% HCl, 2% sodium orthophenylphenate (9) and sodium hypochlorite (1,9).
PHYSICAL INACTIVATION: Inactivated at low pH (1.5)(9) and immediately after heating to 60 °C(10,11). VSV in stroma-free hemoglobin can also be inactivated by phototreatment (e.g., with red light-emitting diode (655 nm), 1,9-dimethylmethylene blue (DMMB), or methylene blue (MB))(11).
Most human infections with VSV
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